Caratteristica: Esplorando i legami tra SARS-CoV-2, 'vaccini' covidi, HIV e immunodeficienza

18 marzo 2022

Data: 18 marzo 2022

Sezioni di contenuto

  • Svelare l'eredità di Montagnier
  • Eruzione immunitaria da iniezioni di covid-19
  • Motivi dell'HIV nel SARS-CoV-2
  • Confondere le acque torbide con Ad5
  • Autoimmunità da iniezioni di covid-19
  • Taglio al bersaglio sugli inserti per l'HIV
  • Conclusioni
  • Riferimenti

Rob Verkerk PhD, fondatore, direttore esecutivo e scientifico, ANH-Intlco-presidente del Comitato Salute e Umanità, Consiglio Mondiale per la Salute

TOPLINE

  • Perez e il premio Nobel Montagnier hanno identificato 18 sequenze di geni nell'HIV-1 che sono presenti nella proteina spike del SARS-CoV-2.
  • Tra questi c'è il gp120, che facilita l'attaccamento del 'picco' dell'HIV alle cellule dell'ospite e aiuta l'HIV a colpire le cellule T CD4.
  • Le prove emergenti mostrano che l'esposizione cronica ai 'vaccini' covid-19, che si verifica attraverso la somministrazione di richiami regolari, può disturbare le cellule T in generale e, più in particolare, sopprimere le cellule T CD4 che sono bersaglio di gp120.
  • Tale esposizione cronica può anche erodere l'importantissima immunità innata e aumentare il rischio di nuove condizioni autoimmuni. Queste potrebbero contribuire a ciò che è stato descritto come VAIDS (sindrome da immunodeficienza acquisita indotta da vaccino).
  • Nonostante i danni noti per i pazienti affetti da HIV/AIDS, causati da un virus del raffreddore comune geneticamente modificato (adenovirus di tipo 5), utilizzato come vettore nelle sperimentazioni STEP nei primi anni 2000, alcuni produttori di vaccini, con l'approvazione dell'OMS, stanno continuando lo sviluppo preclinico o clinico con questi stessi vettori adenovirus.
  • Alcuni dei motivi dell'HIV presenti nel SARS-CoV-2 sono altamente funzionali in termini di facilitazione dell'attacco e della fusione con le cellule bersaglio dell'ospite, ma mancano nel virus della SARS, geneticamente molto simile.
  • Le persone che sono già compromesse dal punto di vista immunitario o che hanno avuto una storia di cancro dovrebbero valutare molto attentamente i rischi del covid-19 e dei vaccini, così come i benefici. Dovrebbero anche considerare le numerose alternative prima di conformarsi semplicemente a quelle che sono diventate norme sociali, nonostante la comune assenza di prove della necessità medica. 

Svelare l'eredità di Montagnier

Nel febbraio 2020, appena un mese dopo la pubblicazione del genoma del SARS-CoV-2, lo scienziato e matematico francese Jean Claude Perez - e collega del Prof. Luc Montagnier, recentemente scomparso - ha pubblicato un articolo intitolato 'Wuhan Covid-19 Synthetic Origins and Evolution' sul server di preprint ResearchGate.[1]. Il documento è stato pubblicato il mese successivo nella rivista peer-reviewed Giornale internazionale di ricerca.  

Tra i lavori di Perez in silico Le scoperte sono state la presenza di frammenti del genoma di due varianti di due retrovirus, il virus dell'immunodeficienza umana (HIV) e il virus dell'immunodeficienza simion (SIV), nel genoma di riferimento della SARS-CoV-2 proveniente dal mercato ittico di Wuhan. La scoperta della presenza di questi frammenti genetici ha fatto sì che Perez fosse tra i primi a sollevare dubbi nella letteratura scientifica sulla pretesa origine naturale e zoonotica del genoma della SARS-CoV-2. Il suo ragionamento è stato che è improbabile che questi virus abbiano trovato la strada in una grotta di pipistrelli nella Cina remota o, ancora, in un ospite intermedio non identificato che potrebbe aver trovato la strada, vivo o morto, verso il mercato del pesce.

Prof Luc Montagnier, Premio Nobel per la Fisiologia o la Medicina, 2008.

Montagnier, in qualità di co-scopritore dell'HIV, per il quale è stato insignito del Premio Nobel nel 2008, ha continuato a collaborare con Perez in un altro lavoro, anch'esso pubblicato sulla rivista Giornale internazionale di ricercaNel luglio 2020.[2] L'analisi presentata ha fornito ulteriori dettagli sulle scoperte iniziali di Perez. Tra queste, il fatto che 2,5% dell'intero genoma della SARS-CoV-2 'Wuhan' era rappresentato da 18 'inserzioni' di frammenti di RNA provenienti dai retrovirus HIV o SIV, con una sezione che aveva un tasso di densità di questi inserti fino a 73%. Gli autori hanno affermato che, poiché i frammenti avevano una lunghezza compresa tra i 18 e i 30 nucleotidi, avevano la capacità di modificare l'espressione genica negli esseri umani esposti al SARS-CoV-2. Hanno anche proposto che la presenza di questi inserti era probabilmente il risultato di una manipolazione umana, potenzialmente sia per la ricerca sul guadagno di funzioni per migliorare la penetrazione cellulare del virus, ma anche per lo scopo di "progettazione del vaccino".  

Le parole finali del documento - pubblicato a pochi mesi dall'inizio della pandemia - erano rivolte ai presunti architetti della SARS-CoV-2 e fornivano un cupo avvertimento:

Questa analisi, realizzata in silico, è dedicata ai veri autori del Coronavirus COVID-19. Spetta solo a loro descrivere i loro esperimenti e perché si sono trasformati in un disastro mondiale: 650.000 vite umane (il 26 luglio 2020), più di quelle prese dalle due bombe atomiche di Hiroshima e Nagasaki. Noi, i sopravvissuti, dovremmo trarre insegnamento da questo grave allarme per il futuro dell'umanità. Esortiamo i nostri colleghi scienziati e medici a rispettare le regole etiche espresse dal giuramento di Ippocrate: non nuocere, mai e poi mai! [2]

Più recentemente, e poco prima della scomparsa di Montagnier l'8 febbraio 2022, all'età di 85 anni, la seguente citazione attribuita al Premio Nobel è circolata ampiamente su Internet:

"Per coloro che hanno assunto la terza dose, vada a fare il test per l'AIDS. Il risultato potrebbe sorprenderla. Allora faccia causa al suo governo". (ad esempio [3])

Non è stato possibile verificare l'autenticità della citazione, ma, insieme alla scoperta di una nuova variante altamente virulenta dell'HIV nei Paesi Bassi all'inizio di febbraio,[4] la scena è stata allestita per le preoccupazioni del pubblico e di alcuni professionisti della salute sui possibili legami tra l'HIV, le iniezioni di covid-19 e la SARS-CoV-2. 

Eruzione immunitaria da iniezioni di covid-19

A questo si aggiunge la crescente preoccupazione degli scienziati, come il famoso vaccinologo belga Geert Vanden Bossche PhD, che iniezioni successive di covid-19 possano compromettere l'efficacia del sistema immunitario, in particolare l'immunità innata formata a seguito di un'infezione acquisita naturalmente.[5]

Il Dr. Vanden Bossche ha proposto che alti livelli di anticorpi 'vaccinali' non sterilizzanti ('leaky'), prodotti in seguito all'iniezione, sopprimano gli anticorpi polireattivi, tutti importanti, prodotti da sottopopolazioni specializzate di cellule B (cellule B-1 e cellule B della zona marginale) associate al sistema immunitario innato.[6]

 

Un'immagine sul sito web del Dr. Geert Vanden Bossche, che include un messaggio all'OMS

Sebbene l'immunità innata sia la prima linea di difesa per tutti, sono soprattutto i bambini a fare maggiore affidamento su di essa, data l'immaturità del braccio adattativo del loro sistema immunitario, che è il meccanismo primario di difesa contro i patogeni respiratori negli adulti.[7]

L'assenza di qualsiasi motivazione scientifica o medica sostanziale per 'vaccinare' i bambini contro il covid-19 è trattata in modo esauriente da Kostoff et al (2021)[8] e Seneff et al (2022)[9]. Lo scopo delle iniezioni di covid-19 non è ovviamente quello di regolare il sistema immunitario innato, ma piuttosto di neutralizzare gli anticorpi nel braccio adattativo del sistema immunitario (la risposta immunitaria umorale). Pertanto, qualsiasi erosione dell'immunità innata o interruzione dell'immunità adattativa cellulo-mediata (attraverso le cellule T) associata all'esposizione regolare alle iniezioni di covid-19 deve essere considerata un danno collaterale.

Sebbene stiano iniziando ad emergere prove meccanicistiche, cliniche e persino epidemiologiche di tale alterazione del sistema immunitario, potrebbero passare anni prima che l'importanza degli effetti di tale erosione o alterazione su diversi gruppi di popolazione con uno stato di salute variabile sia ampiamente compresa e riconosciuta.

Un altro pezzo emergente del puzzle che collega la potenziale erosione immunitaria con l'HIV è la possibilità di sviluppo della 'sindrome da immunodeficienza acquisita da vaccino' o VAIDS (ad esempio, riferimenti [10] e [11]). Sono stati fatti dei tentativi da parte dei 'fact checker' e dei media tradizionali per sfatare tali affermazioni (ad esempio, riferimenti a [12] e [13]) but these challenges to the existence of VAIDS are scientifically hollow and appear to be politically or economically motivated.

With increasing frequency of exposure of people to covid-19 injections that erode innate immunity and disrupt cell-mediated (T cell) immune responses, it is highly likely we will witness a rise in VAIDS. It may be longer before health authorities and vaccine manufacturers who have pushed to achieve incredibly high rates of vaccine coverage in many industrialised countries are prepared to recognise that the injections are the cause.

The two, interconnected arms of the immune system (Source: Getty) 

Motivi dell'HIV nel SARS-CoV-2

There have been concerted efforts by ‘fact checkers’, among them Associated Press[14] and Reuters[15], to denounce any possible link between the so-called covid-19 ‘vaccines’ and HIV or AIDS. Full Fact, for example, stated on February 4, 2022, that “As Covid-19 vaccines don’t contain HIV, they cannot cause AIDS."[16]

As is so often the case: the devil is in the detail. The ‘fact checkers’ are indeed literally correct given, as shown by Perez and Montagnier[2], the whole genome of HIV (or that of SIV) is absent from SARS-CoV-2. But 18 inserts are clearly present and it was a reasonable assumption by Perez and Montagnier to claim this would be unlikely to arise by chance.

In April 2020, mathematician and IT consultant Philippe Lacoude PhD, writing in the European Scientist, penned what appears at face value to be a killer rebuttal of Perez and Montagnier’s findings[17]. Lacoude had read the paper and heard Montagnier speaking about it on the French CNews channel[18]. So he knew that it was only RNA fragments, not the whole HIV genome, that were being claimed to be present in the surface proteins of both viruses; the whole genome of the reference SARS-CoV-2 genome being given in reference[19].

Lacoude leads the reader by the hand and shows how he’s right and his fellow countryman and Nobel laureate, who knows a thing or two about viral genomes, is wrong. He explains that it would be hard to check this manually given the size of the two genomes, so he suggests automating the process by using the MegaBLAST subprogram in the Basic Local Alignment Search Tool (BLAST) developed by the National Institutes of Health (NIH)[20], which hunts down common sequences in different genomes. The long and short is the program fails to detect all 18 of the HIV-1 fragments found by Perez & Montagnier, including gp120 (I’ll come on to that below).

Time to mention an important scientific premise: a lack of evidence, or an inability to find evidence, of a particular phenomenon, does not mean that the phenomenon does not exist.

Confondere le acque torbide con Ad5

Another linkage between HIV and Covid-19 injections is the fact that two ‘vaccines’ already in clinical use (CanSino Biologics and Sputnik), as well as several in the preclinical development phase including one that is delivered orally, utilise Merck’s controversial genetically engineered (GE) adenovirus type-5 vector (Ad5)[21]. This GE common cold virus shuttles the gene for the SARS-CoV-2 spike protein into the body.

Four scientists involved with the fateful STEP trial that was intended to be a proof-of-concept for an HIV vaccine in the 2000s sounded a note of caution on the use of Ad5 in covid-19 ‘vaccines’[22]. The trial relied on Ad5 to vector the gene for the surface protein of HIV and ended up increasing HIV infection risk among vaccinated compared with unvaccinated men[23]. The scientists’ warning came about because their experiences with the STEP trials meant there was a reasonable scientific basis to be concerned that covid-19 ‘vaccines’ reliant on Ad5 may exacerbate AIDS among those already infected with HIV.

Dr Anthony Fauci, who has headed the National Institute of Allergy and Infectious Diseases (NIAID) at the NIH since 1984, went on the public record in 2014 recommending against further use of Ad5 in HIV vaccines[24]. Yet they are being used today in covid-19 ‘vaccines’ (search ClinicalTrials.gov), although sometimes with further genetic alterations intended to partially mute their effects on the body.  

Fauci is listed as the inventor of 2 patents assigned to the Department of Health and Human Services for HIV vaccines that rely on preventing Gp120 from binding to the alpha4 integrin receptor. These are shown in reference [25] and reference [26].

Autoimmunità da iniezioni di covid-19

When considering the susceptibility of individuals to viruses such as HIV or SARS-CoV-2, and the potential for covid-19 injections – especially if delivered at frequent (e.g. 6-monthly) intervals – to compromise immunity, the risk of autoimmunity must be factored in.  

Autoimmunity from adenoviral vectored covid-19 injections was the first recognised adverse autoimmune phenomenon noted following to mass roll-out of the covi-19 injections in 2020. Fortunately, it is generally considered rare, given it can be lethal.

One estimate from Canada suggests that for the AstraZeneca injection, incidence may on average be as high as 1 case per 26,500 (around 4 cases per 100,000)[27], with mortality among those affected having been estimated at 17% in Australia[28]. The incidence also goes up in younger people[29].

In many industrialised countries, the mRNA injections are being more commonly administered as boosters and to younger people, given the known acute (Guillain-Barré syndrome) and chronic (vaccine-induced immune thrombotic thrombocytopenia [VITT], cerebral venous sinus thrombosis)[30] autoimmunity risks from adenoviral vectored injections.

Unfortunately, emerging evidence for mRNA ‘vaccines’ inducing autoimmunity is now emerging. This has been shown clearly for new-onset autoimmune hepatitis, with the suggestion that the injections may trigger inflammatory cascades and autoreactive lymphocytes in susceptible individuals[31].

In addition, as the timeline since the mass injection programs were commenced marches on, there are an increasing number of reports emerging over new-onset autoimmune phenomena that are initiated often days after injection, including autoimmune liver diseases, Guillain-Barré syndrome, IgA nephropathy, rheumatoid arthritis and systemic lupus erythematosus[32].

Covid-19 injections must therefore be considered not only as medical interventions that deliver a potential, often over-stated benefit in terms of protection against risk of severe disease, they also present a potential trigger for unpredictable, long-term, life-changing or even lethal autoimmune conditions.

Taglio al bersaglio sugli inserti per l'HIV

An undeniable scientific tenet is that the smaller the fragment of a given genome identified, the more widely that fragment will be found in the genome of other animal, plant or microbial species.

Let’s now be more specific. It’s been estimated that 87% of the sequence of the HIV-1 glycoprotein envelope and the SARS-CoV-2 spike protein are shared. HIV-1 is a lentivirus, while SARS-CoV-2 is a beta-coronavirus – so they are not closely related despite both being RNA viruses[33]. It could be argued that these similarities are the result of the proteins performing much the same job: being covered in host-derived glycans (carbohydrates) that serve as the basis of “glyco-epitope mediated cross-reactivity by antibodies”,[34] helping each viral particle to fuse with its respective host, facilitating entry of its precious RNA cargo so that viral replication can begin in earnest. Both viruses have evolved to do this well – hence the profound impacts each has had on human populations.

Encoded in the heart of the HIV-1 genome is a long protein called gp160 (gp is short for glycoprotein). This protein is critical to the fusion process. As the gp160 envelope protein fuses with the host cell surface, it cleaves into two distinct pieces, these being gp120 and gp41 respectively. Three gp120 and gp41s then combine in a ‘trimer’ of ‘heterodimers’ to form the envelope spike that locates, attaches to and fuses with the host cell. In the case of HIV-1, attachment occurs via CD4 receptors on these lymphocytes (T cells)[35].

 

Yes, you’ve guessed it, gp120 is also encoded into the SARS-CoV-2 spike protein. It would be easy to dismiss this as an evolutionary trait shared by the two unrelated DNA viruses that occurred by chance or through natural selection. But consider for a minute that the closely related SARS coronavirus that was responsible for the outbreak that was discovered in Asia in February 2003, lacks the gp120 and Gag inserts shared by HIV-1 and SARS-CoV-2. Or that pressure was placed on Indian scientists to withdraw a paper published on a preprint server on January 31, 2020, because it made this connection[36].

This kind of genetic manipulation happens to also be exactly the kind of thing that is done in gain-of-function research, for example to facilitate entry of a manipulated coronavirus to its host. In fact, it is the very sort of genetic engineering that molecular biologist and immunologist Ralph Baric, and colleagues of his ilk, engage with during their work hours[37]. Dr Baric, you may remember, was the NIH-funded scientist who found himself at the centre of controversies around gain-of-function research and possible lab manipulation of SARS-CoV-2 during the early part of the pandemic because of his lab’s long standing research on engineered coronaviruses[38].

Given the similarities in some of the protein motifs in these two unrelated viruses, HIV and SARS-CoV-2, as well as commonalities in the corresponding carbohydrates that cover their surfaces (hence them being referred to as glycosylated proteins), it is of interest that cross-reactive, broadly neutralizing antibodies generated by HIV can also bind to the glycosylated spike protein of SARS-CoV-2[39]. This might suggest HIV positive, asymptomatic individuals could even be at an advantage to their non-HIV exposed counterparts if exposed to circulating SARS-CoV-2 as cross-reactive T cells might be ready for business when confronted with SARS-CoV-2-infected cells.

But it also means that the presence of the gp120 protein in the SARS-CoV-2 spike could help the coronavirus, or the very similar (but not molecularly identical) spike protein produced following covid-19 injection, target T cells, so knocking out all important, multi-function CD4 T cells that have the capacity to differentiate into an array of different subtypes that can provide long-term memory of previous antigens and kill off infected cells. This CD4 lymphocytopenia is obviously one of the hallmarks of HIV positive individuals who go on to develop AIDS.

It is now well recognised from observational studies and autopsy data that reduced CD4 and CD8 T cell counts (lymphopenia) is a key feature of severe covid-19 disease[40]. However, a study published in March 2022 in the journal Signal Transduction and Targeted Therapy, co-authored by Shi Zheng-Li, the so-called ‘bat women’ scientist from the Wuhan Institute of Virology, showed that SARS-CoV-2 targets CD4 and CD8 T cells independently of their infection via ACE2 receptors. This can lead to catastrophic T cell death (apoptosis), with potentially even a zero T cell count in the most severely affected individuals[41].

Conversely, people who experience mild disease and rapid clearance of SARS-CoV-2 have been shown to mount a marked T cell response[42], although a partially effective innate immune response is a likely major contributory factor in preventing severe disease or death.  

A person living with HIV with already compromised T cell immunity courtesy of the virus, would also be expected to not fair well with repeated co-infection with SARS-CoV2 or exposure to covid-19 injections.  A case report from China, involving a 41-year-old patient injected with the Sinopharma inactivated covid-19 ‘vaccine’ showed a dramatic drop in CD4 count[43]. Such T cell disruption is also likely to increase risk of tumour formation particularly among individuals with a history of cancer, a disturbing trait that is already being reported anecdotally by clinicians. Sadly, what we see today could well be just the tip of the iceberg. 

Conclusioni

Where does this exploration leave us?

Let me to attempt to summarise:

  • SARS-CoV-2 is likely a lab construct. There is incontrovertible evidence that there are highly functional genetic fragments that facilitate viral entry and targeting of T cells shared between HIV-1, the main variant of HIV that contributes to AIDS, and SARS-CoV-2. The fact that SARS-CoV-2 is closely related to SARS (and yet doesn’t share the gp120 or Gag sequences that are present in HIV-1 is of particular interest. While there is insufficient evidence to prove that these inserts are definitely the result of gain-of-function research, there is ample evidence it was ongoing in the Wuhan Institute of Virology that was supported by the NIH, despite Anthony Fauci’s denial to the contrary to Congress[44]. This implies a reasonable possibility that these inserts, as Prof Montagnier and others had figured, were likely deliberately inserted and that SARS-CoV-2 is at least in part a lab construct.
  • Chronic exposure to covid-19 vaccines can impair immune function over time. Exposure to successive covid-19 ‘vaccines’ may cause chronic damage to the function of the immune system, notably through the erosion of innate immunity and disruption of T cell responses. Additionally, they may induce autoimmunity and increase the risk of new-onset autoimmune conditions, although the delay and complexity of these conditions mean it might take years to fully understand the scope of disturbance caused. As with any environmental trigger or toxin, it is the dose that makes the poison, as Swiss physician and chemist Paracelsus asserted nearly 500 years ago, so greater frequency or number of exposures to covid-19 injections may induce a dose-response and increased disruption of immune processes.
  •  


    “Those who’ve yet to see what nature has to offer when we’re confronted with unpredicted existential threats, seem unable to see the wood for the trees. Or they have vested reasons that make them persist with failing and often harmful new-to-nature technologies.” - Rob Verkerk PhD

     

  • VAIDS is a thing. There is emerging evidence of the existence of a form of vaccine-induced immune suppression that could be referred to as VAIDS, although the mechanisms may be variable between individuals and are still not clear. Among them is innate immune erosion, T cell disturbances and autoimmunity, but there may also be specific targeting of CD4 T cells by the gp120 insert in the SARS-CoV-2 spike protein. This may even be of greater concern in the case of covid-19 mRNA and adenoviral vector ‘vaccines’ that generated spike protein within the body which may then be exposed over weeks if not months.
  • Particular caution must be exercised for those with compromised immune systems. A significant proportion of those living with HIV suffer CD4 suppression (lymphopenia) and the balance of risk versus benefit should be carefully considered along with informed consent before covid-19 injections are recommended for this, or other immune-suppressed, population groups. Among the factors for consideration are the duration of exposure to spike protein in the event of naturally-acquired infection versus following administration of covid-19 ‘vaccines’, as well as the risk posed by the circulating variant when appropriate measures are taken. These include the use of safe, early treatment protocols (e.g. those developed by the Front Line Covid-19 Critical Care Alliance[45]) as an alternative to covid-19 vaccines that currently do little or nothing to stop transmission and protect against severe disease or death for a few weeks at most, encouraging chronic administration with its consequent problems.      
  • Ultimately, nature takes its course, and it is interesting to posit how nature has fared against human technology in the form of synthetic biology ‘genetic vaccines’ and new-to-nature therapeutics. Human technology has delivered very little for massive investment and cost to society.

    Compare that with our natural protection against SARS-CoV-2, comprised of our incredibly sophisticated immune systems when amply resourced by products of nature, be these healthy foods or specific nutrients, plant or microbial extracts. This is the natural system of defence that got us this far, and it’s been doing its best to cope with, and adapt to, the rapidly changing spectre of this complex and provoked relationship that kicked off less than 3 years ago.

    Those who’ve yet to see what nature has to offer when we’re confronted with unpredicted existential threats, seem unable to see the wood for the trees. Or they have vested reasons that make them persist with failing and often harmful new-to-nature technologies.

     

    For more information: search the Alliance for Natural Health International and World Council for Health websites.

    Riferimenti

    [1] Perez, J-C. Wuhan covid-19 synthetic origins and evolution. Int J Res, 2020; 8(2): 285-3324.

    [2] Perez J-C, Montagnier L. Covid-19, SARS and bat coronavirus genomes peculiar homologous RNA sequences. Int J Res. 2020; 8(07): 2394-3629.

    [3] Reddit: https://www.reddit.com/r/censoredreality/comments/srah00/dr_luc_montagnier_the_doctor_who_discovered_the/

    [4] University of Oxford News. ‘New highly virulent and damaging HIV variant discovered in the Netherlands’, February 4, 2022.

    [5] Q&A with Geert Vanden Bossche #1, ‘From an immunological point of view, boosters and mass vaccination are as harmful as mold on a wall’, December 1, 2021.

    [6] Lee J, Choi J, Suh, J-Y. Establishment of B-1 cell-derived polyreactive monoclonal antibodies and expression of costimulators by B-cell to antigenic stimulation. J Kor Acad Periodon, 2007; 337: 371-384.

    [7] Verhoeven D. Immunometabolism and innate immunity in the context of immunological maturation and respiratory pathogens in young children. J Leukoc Biol. 2019; 106(2): 301-308.

    [8] Kostoff RN, Calina D, Kanduc D, et al. Why are we vaccinating children against COVID-19? [published correction appears in Toxicol Rep. 2021; 8: 1981]. Toxicol Rep. 2021; 8: 1665-1684.

    [9] Seneff S, Nigh G, Kyriakopoulos AM, et al. Innate Immune Suppression by SARS-CoV-2 mRNA Vaccinations: The role of G-quadruplexes, exosomes and microRNAs. Authorea. January 21, 2022. DOI: 10.22541/au.164276411.10570847/v1.

    [10] AmericasFrontlineDoctors.org. ‘Vaccine Acquired Immune Deficiency Syndrome (VAIDS): ‘We should anticipate seeing this immune erosion more widely’’, December 6, 2021.

    [11] The New Moral Order. “The ‘VAIDS’ PANDEMIC (Vaccine Acquired Immunodeficiency Syndrome)”, November 23, 2021.

    [12] Reuters fact Check. Fact Check-‘VAIDS’ is not a real vaccine-induced syndrome, experts say; no evidence COVID-19 vaccines cause immunodeficiency. February 11, 2022

    [13] Euronews. “Debunked: COVID-19 vaccines do not create variants of HIV/AIDS”, February 10, 2022

    [14] AP News. “Virulent HIV variant is decades old, has no link to COVID vaccine”, February 7, 2022

    [15] Reuters. “Fact Check-COVID-19 vaccines do not cause HIV or AIDS”, February 2021

    [16] Full Fact. “There is no HIV in covid-19 vaccines.”, October 15, 2021

    [17] Lacoude, P. ‘No, SARS-CoV-2 does not contain HIV genetic code!’ EuropeanScientist.com, April 18, 2020.

    [18] CNews tweet, April 17, 2020

    [19] SARS-CoV-2 Sequence Resources, NIH/National Library of Medicine.

    [20] Basic Local Alignment Search Tool

    [21] WHO COVID-10 vaccine tracker and landscape. Searched 15 March 2022.

    [22] Buchbinder SP, McElrath MJ, Dieffenbach C, Corey L. Use of adenovirus type-5 vectored vaccines: a cautionary tale. Lancet. 2020 Oct 31;396(10260):e68-e69.

    [23] Sekaly RP. The failed HIV Merck vaccine study: a step back or a launching point for future vaccine development?. J Exp Med. 2008; 205(1): 7-12.

    [24] Fauci AS, Marovich MA, Dieffenbach CW, Hunter E, Buchbinder SP. Immunology. Immune activation with HIV vaccines. Science. 2014; 344(6179): 49-51.

    [26] Google patent: Use of antagonists of the interaction between HIV GP120 and α4β7 integrin.

    [27] Chan B, Odutayo A, Juni P et al. Risk of vaccine‐induced thrombotic thrombocytopenia (VITT) following the AstraZeneca/COVISHIELD adenovirus vector COVID‐19 vaccines. Science Briefs of the Ontario COVID‐19 Science Advisory Table. 2021; 2. 10.47326/ocsat.2021.02.28.1.0.

    [28] Australian Technical Advisory Group on Immunisation (ATAGI). ATAGI update following weekly COVID‐19 meeting – 18 August 2021. 2021. [Accessed March 16, 2022].

    [29] McLean-Tooke A, Michaela L, French M. Autoimmunity elicited by the chemokine response to adenovirus vector vaccines may underlie vaccine-induced immune thrombotic thrombocytopaenia: a hypothesis. Clin Transl Immunol. 2021; 10(10): e1349.

    [30] Franchini M, Liumbruno GM, Pezzo M. COVID-19 vaccine-associated immune thrombosis and thrombocytopenia (VITT): Diagnostic and therapeutic recommendations for a new syndrome. Eur J Haematol. 2021 Aug;107(2):173-180.

    [31] McShane C, Kiat C, Rigby J, Crosbie Ó. The mRNA COVID-19 vaccine – A rare trigger of autoimmune hepatitis? J Hepatol. 2021;75(5):1252-1254. doi:10.1016/j.

    [32] Chen Y, Xu Z, Wang P, Li XM, Shuai ZW, Ye DQ, Pan HF. New-onset autoimmune phenomena post-COVID-19 vaccination. Immunology. 2021 Dec 27. doi: 10.1111/imm.13443.

    [33] Fischer W, Giorgi EE, Chakraborty S, et al. HIV-1 and SARS-CoV-2: Patterns in the evolution of two pandemic pathogens. Cell Host Microbe. 2021;29(7):1093-1110.

    [34] Mannar D., Leopold K, Subramaniam S. Glycan reactive anti-HIV-1 antibodies bind the SARS-CoV-2 spike protein but do not block viral entry. Sci Rep, 2011; 11: 12448.

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